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This study was performed to investigate the effects of exopolysaccharide and lipopolysaccharide lipoprotein complexes (LPS-LPC) on protein phosphorylation in rat lung. In 5-day-old rats, administration of LPS-LPC into the trachea caused a significant increase in protein kinase C (PKC) activity in the lung; protein kinase A activity was not affected. In 4- and 10-day-old rats, the levels of PKC activity in the lung increased after LPS-LPC administration. In these cases, LPS-LPC administration also induced an increase in PKC activity in a lung microsomal fraction
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